ER stress in prostate cancer: A therapeutically exploitable vulnerability?

Research output: Contribution to journalArticlepeer-review

8 Scopus citations

Abstract

Cooperative oncogenic effects resulting from the loss of PTEN and overexpression of MYC overcome the deleterious effects of endoplasmic reticulum stress not only to promote the growth of aggressive prostate cancer but also to expose a new therapy target for this disease (Nguyen et al., this issue).

Original languageEnglish (US)
Article numbereaat3975
JournalScience translational medicine
Volume10
Issue number439
DOIs
StatePublished - May 2 2018

ASJC Scopus subject areas

  • General Medicine

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