Abstract
In this issue of Blood, Kasirer-Friede and colleagues show that ADAP is a component of a signaling system triggered when blood flow pulls αIIbβ3 bound to fibrinogen. It serves to convert tension into a biochemical response that stabilizes platelet attachment by directing lamellipodia formation.
Original language | English (US) |
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Pages (from-to) | 2123-2124 |
Number of pages | 2 |
Journal | Blood |
Volume | 115 |
Issue number | 11 |
DOIs |
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State | Published - Mar 18 2010 |
ASJC Scopus subject areas
- Biochemistry
- Immunology
- Hematology
- Cell Biology