Sugar? No thank you, just a deep breath of oxygen for cancer stem cells

Andrea Viale; Giulio F. Draetta

doi:10.1016/j.cmet.2015.09.020

Sugar? No thank you, just a deep breath of oxygen for cancer stem cells

Andrea Viale, Giulio F. Draetta

Genomic Medicine

Research output: Contribution to journal › Short survey › peer-review

9 Scopus citations

Abstract

Tumors are metabolically heterogeneous, and subpopulations of tumorigenic cells have been recently described to rely more on mitochondrial respiration than glycolysis for energy production. In this issue, Sancho et al. (2015) demonstrate that MYC is a master switch regulating metabolic programs in different subpopulations of pancreatic tumor cells.

Original language	English (US)
Pages (from-to)	543-545
Number of pages	3
Journal	Cell Metabolism
Volume	22
Issue number	4
DOIs	https://doi.org/10.1016/j.cmet.2015.09.020
State	Published - Oct 6 2015

ASJC Scopus subject areas

Physiology
Molecular Biology
Cell Biology

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10.1016/j.cmet.2015.09.020

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title = "Sugar? No thank you, just a deep breath of oxygen for cancer stem cells",

abstract = "Tumors are metabolically heterogeneous, and subpopulations of tumorigenic cells have been recently described to rely more on mitochondrial respiration than glycolysis for energy production. In this issue, Sancho et al. (2015) demonstrate that MYC is a master switch regulating metabolic programs in different subpopulations of pancreatic tumor cells.",

author = "Andrea Viale and Draetta, {Giulio F.}",

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T1 - Sugar? No thank you, just a deep breath of oxygen for cancer stem cells

AU - Viale, Andrea

AU - Draetta, Giulio F.

PY - 2015/10/6

Y1 - 2015/10/6

N2 - Tumors are metabolically heterogeneous, and subpopulations of tumorigenic cells have been recently described to rely more on mitochondrial respiration than glycolysis for energy production. In this issue, Sancho et al. (2015) demonstrate that MYC is a master switch regulating metabolic programs in different subpopulations of pancreatic tumor cells.

AB - Tumors are metabolically heterogeneous, and subpopulations of tumorigenic cells have been recently described to rely more on mitochondrial respiration than glycolysis for energy production. In this issue, Sancho et al. (2015) demonstrate that MYC is a master switch regulating metabolic programs in different subpopulations of pancreatic tumor cells.

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DO - 10.1016/j.cmet.2015.09.020

M3 - Short survey

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JO - Cell Metabolism

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ER -

Sugar? No thank you, just a deep breath of oxygen for cancer stem cells

Abstract

ASJC Scopus subject areas

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