Abstract
The tumor suppressor protein 53BP1 decorates DNA damage sites and is instrumental for nonhomologous end joining. Evidence that 53BP1 facilitates synapsis of DNA ends by modulating chromatin dynamics reveals a hitherto unanticipated strategy for joining distant ends.
Original language | English (US) |
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Pages (from-to) | 1241-1242 |
Number of pages | 2 |
Journal | Nature Structural and Molecular Biology |
Volume | 15 |
Issue number | 12 |
DOIs | |
State | Published - Dec 2008 |
Externally published | Yes |
ASJC Scopus subject areas
- Structural Biology
- Molecular Biology