Akt-mediated YB-1 phosphorylation activates translation of silent mRNA species

Valentina Evdokimova, Peter Ruzanov, Michael S. Anglesio, Alexey V. Sorokin, Lev P. Ovchinnikov, Jonathan Buckley, Timothy J. Triche, Nahum Sonenberg, Poul H.B. Sorensen

Research output: Contribution to journalArticlepeer-review

221 Scopus citations

Abstract

YB-1 is a broad-specificity RNA-binding protein that is involved in regulation of mRNA transcription, splicing, translation, and stability. In both germinal and somatic cells, YB-1 and related proteins are major components of translationally inactive messenger ribonucleoprotein particles (mRNPs) and are mainly responsible for storage of mRNAs in a silent state. However, mechanisms regulating the repressor activity of YB-1 are not well understood. Here we demonstrate that association of YB-1 with the capped 5′ terminus of the mRNA is regulated via phosphorylation by the serine/threonine protein kinase Akt. In contrast to its non-phosphorylated form, phosphorylated YB-1 fails to inhibit cap-dependent but not internal ribosome entry site-dependent translation of a reporter mRNA in vitro. We also show that similar to YB-1, Akt is associated with inactive mRNPs and that activated Akt may relieve translational repression of the YB-1-bound mRNAs. Using Affymetrix microarrays, we found that many of the YB-1-associated messages encode stress- and growth-related proteins, raising the intriguing possibility that Akt-mediated YB-1 phosphorylation could, in part, increase production of proteins regulating cell proliferation, oncogenic transformation, and stress response.

Original languageEnglish (US)
Pages (from-to)277-292
Number of pages16
JournalMolecular and cellular biology
Volume26
Issue number1
DOIs
StatePublished - Jan 2006
Externally publishedYes

ASJC Scopus subject areas

  • Molecular Biology
  • Cell Biology

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