New roles opined for OPCML

Sherry Y. Wu; Anil K. Sood

doi:10.1158/2159-8290.CD-11-0356

New roles opined for OPCML

Sherry Y. Wu, Anil K. Sood

Gynecological Oncology & Reproductive Medicine

Research output: Contribution to journal › Article › peer-review

10 Scopus citations

Abstract

OPCML, frequently inactivated in ovarian tumors, mediates its antitumor effect via binding to the extracellular domains of several important oncogenic receptor tyrosine kinases (RTK). This, in turn, leads to the downregulation of RTKs in tumor cells and results in significant inhibition of tumor growth.

Original language	English (US)
Pages (from-to)	115-116
Number of pages	2
Journal	Cancer discovery
Volume	2
Issue number	2
DOIs	https://doi.org/10.1158/2159-8290.CD-11-0356
State	Published - Feb 2012

ASJC Scopus subject areas

Oncology

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10.1158/2159-8290.CD-11-0356

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title = "New roles opined for OPCML",

abstract = "OPCML, frequently inactivated in ovarian tumors, mediates its antitumor effect via binding to the extracellular domains of several important oncogenic receptor tyrosine kinases (RTK). This, in turn, leads to the downregulation of RTKs in tumor cells and results in significant inhibition of tumor growth.",

author = "Wu, {Sherry Y.} and Sood, {Anil K.}",

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New roles opined for OPCML

Abstract

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