STAT3: The "achilles" heel for AML?

Katayoun Rezvani, John Barrett

Research output: Contribution to journalReview articlepeer-review

9 Scopus citations

Abstract

In this issue of Blood, in a series of elegant studies in a transplantable mouse model of acute myeloid leukemia (AML), Hossain et al1 show that systemic signal transducer and activator of transcription 3 (STAT3) blocking/TLR9 triggering can eradicate established AML through an immune-mediated mechanism.

Original languageEnglish (US)
Pages (from-to)1-2
Number of pages2
JournalBlood
Volume123
Issue number1
DOIs
StatePublished - Jan 2 2014

ASJC Scopus subject areas

  • Biochemistry
  • Immunology
  • Hematology
  • Cell Biology

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