Abstract
The analysis of compound mouse mutants for nonhomologous end-joining DNA double-strand break repair and those deficient for the p53 checkpoint pathway has provided a fascinating look at the carcinogenic consequences of the failure to properly repair DNA damage and to elicit appropriate checkpoints.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 4-6 |
| Number of pages | 3 |
| Journal | Cancer cell |
| Volume | 3 |
| Issue number | 1 |
| DOIs | |
| State | Published - Jan 2003 |
| Externally published | Yes |
ASJC Scopus subject areas
- Oncology
- Cell Biology
- Cancer Research