Author Correction: HIC2 controls developmental hemoglobin switching by repressing BCL11A transcription (Nature Genetics, (2022), 54, 9, (1417-1426), 10.1038/s41588-022-01152-6)

Peng Huang, Scott A. Peslak, Ren Ren, Eugene Khandros, Kunhua Qin, Cheryl A. Keller, Belinda Giardine, Henry W. Bell, Xianjiang Lan, Malini Sharma, John R. Horton, Osheiza Abdulmalik, Stella T. Chou, Junwei Shi, Merlin Crossley, Ross C. Hardison, Xiaodong Cheng, Gerd A. Blobel

Research output: Contribution to journalComment/debatepeer-review

Abstract

Correction to: Nature Genetics. Published online 8 August 2022. In the version of this article originally published, the sixth paragraph of the Discussion did not include the following text: “Moreover, Dykes et al.47 [J. Mol. Cell. Cardiol. 114, 29–37 (2018)] presented preliminary evidence that in primitive murine erythroid cells HIC2 may repress the premature activation of definitive type hemoglobin, in agreement with our findings in human cells.” The error has been corrected in the HTML and PDF versions of the article.

Original languageEnglish (US)
Pages (from-to)1608
Number of pages1
JournalNature Genetics
Volume55
Issue number9
DOIs
StatePublished - Sep 2023

ASJC Scopus subject areas

  • Genetics

Fingerprint

Dive into the research topics of 'Author Correction: HIC2 controls developmental hemoglobin switching by repressing BCL11A transcription (Nature Genetics, (2022), 54, 9, (1417-1426), 10.1038/s41588-022-01152-6)'. Together they form a unique fingerprint.

Cite this